A transgenic mouse assay for agouti protein activity.
Characterization of plasma lipids in genetically obese mice: Pulmonary fibrosis caused by N-methyl-N-nitrosourethane inhibits lung tumorigenesis by urethane in transgenic mice carrying the human prototype c-Ha-ras gene. VGF ablation blocks the development of hyperinsulinemia and hyperglycemia in several mouse models of obesity.
Antiobesity and antidiabetic actions of a new potent disaccharidase inhibitor in genetically obese-diabetic mice, KKA y. Menin controls growth of pancreatic beta-cells in pregnant mice and promotes gestational diabetes mellitus. Molecular analysis of reverse mutations from nonagouti a to black-and-tan a t and white-bellied agouti Aw reveals alternative forms of agouti transcripts.
Description of agouti and albino series of allelomorphs Analysis of pancreatic islet cells and hormone content in the spontaneously diabetic KKAy mouse by morphometry, immunocytochemistry and radioimmunoassay. The yellow mouse obesity syndrome and mechanisms of agouti-induced obesity.
Pleiotropic effects of the mouse lethal yellow Ay mutation explained by deletion of a maternally expressed gene and the simultaneous production of agouti fusion RNAs. Fibroblast growth factor receptor 1 is required for the proliferation of hippocampal progenitor cells and for hippocampal growth in mouse.
Genetic organization of the agouti region of the mouse. Mahogany hypotéka s půjčkou stimulates feeding and increases basal metabolic rate independent of its suppression of agouti.
Normalization of prandial blood glucose and improvement of glucose tolerance by liver-specific inhibition of SH2 domain containing inositol phosphatase 2 SHIP2 in diabetic Cofidis předschválená půjčka xl mice: Identification of SOCS-3 as a potential mediator of central leptin resistance. Mutations in mice that influence natural killer NK cell activity. Hereditary diabetes in the KK mouse: Genetics of the early mouse embryo.
Cloning of the mouse agouti gene predicts a secreted protein ubiquitously expressed in mice carrying the lethal yellow mutation. Role of melanocortinergic neurons in feeding and the agouti obesity syndrome [see comments] Differential expression of a new dominant agouti allele Aiapy is correlated with methylation state and is influenced by parental lineage.
Microvasculature of hydronephrotic kidneys in KK-A Y mice. Coincidence of loci for glucosuria and obesity in type 2 diabetes-prone KK-Ay mice. Influence of background genome on enzymatic characteristics of yellow A v - A vy - mice. Ablation of neurons expressing agouti-related protein activates fos and gliosis in postsynaptic target regions.
Oral administration of a zinc complex improves type 2 diabetes and metabolic syndromes. Developmental interaction in the pigmentary system of mice. The agouti suppressor As coat color mutation in mice: Overexpression of pituitary adenylate cyclase-activating polypeptide in islets inhibits hyperinsulinemia and islet hyperplasia in agouti yellow mice. The A y allele at the agouti locus enhances sensitivity to endotoxin-induced lethality in mice.
Pigmentation phenotypes of variant extension locus alleles result from point mutations that alter MSH receptor function.
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